Program
14:00-14:40 Inge Marie Svane “Reaching beyond immune checkpoint inhibitors to defeat metastatic melanoma”
14:40-15:20 Trude Helen Flo “"Inflammatory cell death of human macrophages infected with Mycobacterium tuberculosis"
The lecturers are visiting Oslo as opponents of Maria Stadheim Eggebø’s PhD defence May 28.
Biosketches
Inge Marie Svane, MD, PhD is Professor in Clinical Cancer Immune Therapy at the University of Copenhagen, Director of National Center for Cancer Immune Therapy (CCIT-DK), Herlev and Gentofte Hospital, and Chief Physician specialized in melanoma at the Department of Oncology at Copenhagen University Hospital, Herlev, Denmark.
Inge Marie Svane has more than 25 years of research experience in cancer immunology and immunotherapy and holds a PhD in basic cancer immunology. She has pioneered the field of cancer immunotherapy in Denmark, building up CCIT-DK, a translational research center in a leading European position. Her focus of expertise is T-cell based adoptive cell therapy and other cellular therapies, cancer vaccines, and immune regulation.
Since 2001 she has initiated more than 30 phase I, II, and III trials as academic sponsor within the area of immunotherapy.
Inge Marie Svane is co-author of more than 350 scientific publications. ORCiD: 0000-0002-9451-6037
Trude Helen Flo is a Professor in Cell Biology at NTNU, former Co-Director and Director of the CoE CEMIR: Centre of Molecular Inflammation Research (2013-2024). Flo heads a research group focused on host-pathogen interactions, inflammatory responses and cell death - especially during infection with Mycobacterium tuberculosis, but also other bacterial and viral pathogens. Her team use microscopy to study intracellular trafficking, compartmentalized receptor signaling and pathogen-induced host-cell death modalities, and recently established protocols for genetic editing of human induced pluripotent stem cells (iPSCs) that are used to generate macrophages, alveolar epithelial cells and a co-culture model to study how cellular crosstalk shapes the inflammatory response and infection outcome.